Schizophrenia is a group of unknown etiology of severe mental illness. The symptoms of Schizophrenia are mostly first found on young adults and often manifest in different syndromes involving perception, thinking, feeling and behavior more in and uncoordinated mental activity. Patients are usually conscious and normal in intelligence, but some patients in the disease process would show the damages of cognitive function. The general course of persistent was repeated seizures, aggravation or deterioration in some patients eventually decline and mental disabilities, but some patients may remain cured or substantially cured after treatment.
Scientists have discovered a gene that makes the human's rate of suffering from schizophrenia. What's more important, they also found a biological explanation for this phenomenon.
The main culprit is C4 gene. This gene is associated with the immune system and will need to label the foreign cells. A new study recently published in the journal Nature shows that this gene has a long form, and especially those people with multiple long copies are more likely to develop schizophrenia.
In the test carried out in mice will connect the dots, showing C4 neurons perform the task and what it does for similar foreign cells: produce the synapse marked "trimmed" of proteins. So, if the finding applies to humans, it may explain an intriguing fact: people afflicted by schizophrenia have an abnormally low number of synapses.
Related reading>> http://www.cusabio.com/Polyclonal-Antibody/CD34-Polyclonal-Antibody-11106189.html
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